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The APE1 inefficiently cleaves but recruits various transcription factors to activate downstream genes, such as vascular endothelial growth factor (VEGF) and endonuclease III‐like protein 1 (NTHL1) [95]

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This may relate to increased hydrostatic pressure that occurs during fetal development as systemic blood pressure rises in parallel with plasma protein concentration that provides the “colloid” osmotic pressure important for fluid exchange across capillaries